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Science 11 October 1996:
Vol. 274. no. 5285, pp. 174 - 175
DOI: 10.1126/science.274.5285.174
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News & Comment
Pallava Bagla, Jocelyn Kaiser
New Delhi--A potentially devastating health crisis is quietly unfolding in West Bengal, India, where high levels of arsenic have leached into well water and have put more than a million Indians at risk. The immense scale of the human tragedy has begun to attract the attention of scientists from around the world, who see in this grim event an unprecedented chance to learn more about the health effects of arsenic, a known carcinogen. "We're talking about numbers [of arsenic-poisoning victims] that have never been seen before in the world," says University of California, Berkeley, epidemiologist Allan Smith.
THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
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- K. Yoshida, K. Inoue, Y. Takahashi, S. Ueda, K. Isoda, K. Yagi, and I. Maeda (2008)
Appl. Envir. Microbiol.
74, 6730-6738
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- The geochemical and isotopic composition of ground waters in West Bengal: tracing ground-surface water interaction and its role in arsenic release.
- M. Lawson, C. J. Ballentine, D. A. Polya, A. J. Boyce, D. Mondal, D. Chatterjee, S. Majumder, and A. Biswas (2008)
Mineralogical Magazine
72, 441-444
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- Pregnancy Outcomes, Infant Mortality, and Arsenic in Drinking Water in West Bengal, India.
- O. S. von Ehrenstein, D. N. Guha Mazumder, M. Hira-Smith, N. Ghosh, Y. Yuan, G. Windham, A. Ghosh, R. Haque, S. Lahiri, D. Kalman, et al. (2006)
Am. J. Epidemiol.
163, 662-669
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- Occurrence of arsenic in ground water in the Choushui River alluvial fan, Taiwan..
- C.-W. Liu, S.-W. Wang, C.-S. Jang, and K.-H. Lin (2006)
J. Environ. Qual.
35, 68-75
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- Decrements in Lung Function Related to Arsenic in Drinking Water in West Bengal, India.
- O. S. von Ehrenstein, D. N. G. Mazumder, Y. Yuan, S. Samanta, J. Balmes, A. Sil, N. Ghosh, M. Hira-Smith, R. Haque, R. Purushothamam, et al. (2005)
Am. J. Epidemiol.
162, 533-541
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- Effects of pH on Metals Precipitation and Sorption: Field Bioremediation and Geochemical Modeling Approaches.
- M.-K. Lee, M.-K. Lee, and J. A. Saunders (2003)
Vadose Zone J.
2, 177-185
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- Tumor Promoter Arsenite Stimulates Histone H3 Phosphoacetylation of Proto-oncogenes c-fos and c-jun Chromatin in Human Diploid Fibroblasts.
- J. Li, M. Gorospe, J. Barnes, and Y. Liu (2003)
J. Biol. Chem.
278, 13183-13191
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- Caveolin-Induced Activation of the Phosphatidylinositol 3-Kinase/Akt Pathway Increases Arsenite Cytotoxicity.
- S. Shack, X.-T. Wang, G. C. Kokkonen, M. Gorospe, D. L. Longo, and N. J. Holbrook (2003)
Mol. Cell. Biol.
23, 2407-2414
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- Transcriptional Induction of MKP-1 in Response to Stress Is Associated with Histone H3 Phosphorylation-Acetylation.
- J. Li, M. Gorospe, D. Hutter, J. Barnes, S. M. Keyse, and Y. Liu (2001)
Mol. Cell. Biol.
21, 8213-8224
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- Utility Maximization as a Solution: Promise, Difficulties, and Impediments.
- J. BARON (1999)
American Behavioral Scientist
42, 1301-1321
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- Hypertension and Arsenic Exposure in Bangladesh.
- M. Rahman, M. Tondel, S. A. Ahmad, I. A. Chowdhury, M. H. Faruquee, and O. Axelson (1999)
Hypertension
33, 74-78
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- Tumor Promoter Arsenite Activates Extracellular Signal-Regulated Kinase through a Signaling Pathway Mediated by Epidermal Growth Factor Receptor and Shc.
- W. Chen, J. L. Martindale, N. J. Holbrook, and Y. Liu (1998)
Mol. Cell. Biol.
18, 5178-5188
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- The MRP2/cMOAT Transporter and Arsenic-Glutathione Complex Formation Are Required for Biliary Excretion of Arsenic.
- S. V. Kala, M. W. Neely, G. Kala, C. I. Prater, D. W. Atwood, J. S. Rice, and M. W. Lieberman (2000)
J. Biol. Chem.
275, 33404-33408
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